Introduction
Affects 30-70 year old (adult) males predominantly.
- Bilateral
- Lesions usually recurrent lasting weeks to months
- CNV can occur at margin of chorioretinopathy
Proposed pathophysiology:
- Due to immune mediated vasculitis with subsequent occlusion of choroidal vessels
Dead Giveaways
Fundus Appearance:
Linear grey-white retinal lesion resembling a coiled snake or medusa head, moving away from the disc.
Lesions will develop pigmentation with chronicity 
The inflammation appears as a very dark hypo-AF zone, with outer retinal layers undergoing atrophy. When active, appears with a hyper-AF border.
OCT Appearance:
Active Lesions show hyper-reflectivity of RPE, EZ and ONL. SRF often present.
Chronic Lesions show outer retinal atrophy, choroidal hypertransmission
Shows the outer retinal atrophy, EZ and RPE disruptions 
From Gunduz G.U. et al. 2020. Shows the active EZ disruptions and hyperreflective ILM and what appears to be a FIPED. This FIPED does not have a homogenous dark appearance and appears to show mixed hyper/hypo-reflective material wtihin. This indicates the likely presence of neovascular material, specifically from the choroid (CNV).
diagnostic features
Symptoms:
Typical of the white dot spectrum, such as blurred vision, scotomas, photopsia, floaters.
Can be asymptomatic
Prognosis:
Recurrent multiple inflammatory episodes
CNV occurs in areas of chorioretinal atrophy